Interpreting an MCG Report

Why Premier Heart's MCG will revolutionize the way people see and do healthcare.

Before we dive into the specifics, I’d like to make the following statements:

The hallmark of the failed legacy of allopathic cardiology is a fundamental lack of innovation in a much-needed bedside tool to DETECT signs of physiological dysfunction in their EARLY stages to enable effective disease reversal and timely prevention. All conventional diagnostic tools detect late-stage diseases, with many treatments often being too little and too late. These legacy phenomena are emblematic of the decades-long systematic technological stagnation of a corrupt, ossified, and obsolete system Peter Thiel has written and lamented for years. We have witnessed and investigated the phenomena firsthand! He has been correct all along.

Premier Heart’s Multifunction Cardiogram (MCG) TM fundamentally solves the systemic problems of lack of accurate, reliable, safe, and effective bedside diagnostic tools for the frontline clinicians serving the population daily under a dying legacy system. Premier Heart, LLC leads medicine into the 21st Century and beyond! The future of medicine is with the paradigm of early detection and monitoring of the signs of physiological dysfunctions from the earliest to the latest stages to discover the root causes to prevent and reverse the progression of the disease process, primarily through learning to live an optimal healthier lifestyle, in a cleaner environment, eat healthier wholesome, nutritious foods devoid of “ultra-processed ‘foods’ and ‘toxic’ chemicals,” AND excessive stressful lifestyles, thus to extend a healthier lifespan and ultimately save lives.

Let’s begin.

MCG TEST RESULTS ANALYSIS

ISCHEMIA RESULTS and the MCG SEVERITY SCORES:

Word of Caution: Thanks to our decades of dedicated, on shoestring budgets, but determined, meticulous empirical data mining to train the world’s first empirical evidence, objective, and reality-based, founded on a set of immutable, and immortal mathematical principals, 100% empirical evidence-driven, and patient-benefit-centric deep machine learning neuro-network to aid diagnosis of heart and brain physiological dysfunctions, with a laser-focus on the biological mitochondrial networks as a system, its dysfunctions leading to chronic (and acute) diseases, we have discovered and defined the digital parameters of 1x10 to 168th power, “Quinquinquagintillion” to mirror the biological mitochondrial system’s (containing 10 x of approximate 70K to 100k trillion mitochondria) of the functioning human heart and brain. MCG detects, quantifies, and monitors myocardial Ischemia or cellular quiescence as a result of ANY cause that leads to supply and demand imbalances physiologically, including but not limited to mitochondrial dysfunctions due to anemia, hypoxia, hypoxemia, insulin resistance, Diabetes, nutrient deficiencies, poor sedentary, stressful lifestyles, environmental toxins such as heavy metals, insecticide, pesticides, eating ultra-processed “foods” such as seed oils, sugar, flours, alcohol, etc., tobacco smoking, air pollution, narcotics, such as Fentanyl, Cocaine, the effects of Cynide, environmental toxins, any root causes disrupting mTOR, AMPK pathways’ normal functions, any root cause leading to all stages of heart failure, coronary artery disease, endothelial dysfunction, microvascular disease, neuroendocrine disorders, acute or chronic exposures to stress, Thyroid dysfunction, medications, general anesthesia, high-altitude-sickness, deep-sea-diving accidents (the Bends), and coronary angiogram visible obstructive coronary artery disease (less than 15% in our database). Thus, do not treat the conditions MCG reported as “invisible” (any vessel less than 7mm on their angiogram) to the naked eyes of interventional cardiologists as “false positive” cases to misdiagnose patients’ conditions. These are the blind spots which lead to misdiagnosis and harm to your patients. Thus, clinicians, please explore the information, help your patients on their journey, and strive to find the root cause(s) to save your patients! Please do not irresponsibly lump your patients into a “Syndrome X” bucket of “No-Men’s-Land” and abandon them. You are their last hope for survival, doctors!

It is critical to understand that we created this report to allow clinicians and their patients to read the output using the language they are accustomed to, except for a few pathophysiological conditions, such as incipient arrhythmias, myocardial compliance, and synchronization, which are unique to our technology. Nevertheless, these “foreign” terms, unavailable in the legacy cardiology conventional reporting, are clinically advantageous to denote how the targeted heart functions to save lives.

Global Ischemia denotes Ischemia of the entire heart, whereas local Ischemia denotes partial heart involvement, nothing more.

Note: when encountering alternating global/local/borderline/none ischemia patterns, one must consider the possibilities of “functional ischemia.” Shockingly, the ALMIGHTY “obstructive coronary ischemia” is NOT the first on the differential diagnosis list, certainly not the NUMBER ONE differential diagnosis at all. We now know that coronary interventions, such as PTCA, Stenting, and the Coronary Bypass Artery Grafting (CABG) procedure, produce zero clinical benefits to patients diagnosed with chronic coronary artery disease. One must consider the rationale behind referring a patient to undergo such procedures.

The eight category icons in the report are self-explanatory.

We build MCG on the promise that the technology can and should detect the earliest possible abnormal cardiovascular energy production biological network (i.e., the biological mitochondrial network), which functions for timely lifestyle reversal and disease prevention. MCG is the only test that could deliver that information.

PATHOLOGICAL CONDITIONS: (For clinicians references and pattern recognition purposes only, not definitive diagnosis)

Cardiomyopathy:

Cardiomyopathy (kahrHeart'smy-OP-uh-thee) is a disease of the heart muscle. It causes the heart to have a more challenging time pumping blood to the rest of the body, leading to heart failure symptoms. There are various types of cardiomyopathy. The main types include dilated, hypertrophic, congenital or acquired HOCM, and restrictive cardiomyopathy, among others.

Potential Fibrillation:

These are potentials of small amplitude and brief time frames resulting from a single heart muscle fiber undetectable by the conventional EKG. Their rhythmicity and morphology identify them as fibrillations. Each potential appears the same, suggesting the same heart fiber discharges the same fiber repetitively. These findings are unique to MCG and impossible for ANY conventional legacy tools to detect.

Atrial Fibrillation:

Atrial fibrillation (A-fib) is an irregular heart rhythm that begins in your heart's upper chambers (atria). Symptoms include fatigue, heart palpitations, shortness of breath, and dizziness. A-fib is one of the most common arrhythmias. MCG detects A-fib unconventionally and A-fib unconventionally and thus can, at times, detect when the conventional EKG fails to detect.

Incipient Ventricular Fibrillation: (Initial early stage)

Ventricular fibrillation is a type of irregular heart rhythm (arrhythmia). During ventricular fibrillation, the lower heart chambers contract in a very rapid and uncoordinated manner. As a result, the heart doesn't pump blood to the rest of the body.

Ventricular fibrillation may require immediate medical attention. It's the most frequent cause of sudden cardiac death.

MCG detects V-fib unconventionally, so it can detect it when the traditional EKG fails to do so. Thus, it can detect it when the conventional EKG fails to detect it unconventionally, so it can detect it when the traditional EKG fails to.

Atrial-Ventricular Fibrillation:

Atrial fibrillation (A-Fib) and ventricular fibrillation (V-Fib) are both types of irregular heartbeats or arrhythmias that can be serious. A-Fib affects the upper chambers, while V-Fib affects the lower chambers. Both conditions can cause an irregular and often fast heart rate, but V-Fib can be more dangerous and immediately life-threatening.

MCG detects A-fib/V-fib combination uniquely and unconventionally, thus can detect when the conventional EKG fails to detect.

Incipient Arrhythmia: (Early stages)

An arrhythmia, or irregular heartbeat, is a problem with the rate or rhythm of your heartbeat. Your heart may beat too quickly, too slowly, or with an irregular rhythm. One expects your heart rate to speed up during physical activity and to slow down while resting or sleeping.

MCG detects Incipient Arrhythmia uniquely and unconventionally, thus can detect when the conventional EKG fails to detect.

Ventricular arrhythmia: (Initial early stage)

Ventricular arrhythmias are abnormal heartbeats in the inner area caused by the lower heart chambers, called ventricles. They cause the heart to beat too fast, which prevents oxygen-rich blood from circulating to the brain and body and may result in cardiac arrest.

Myocardial Damage

When an area of the heart muscle (myocardium) is damaged or dies after the blood supply is blocked, it's the classic medical term for a heart attack.

Other causes of myocardial cellular senescence due to loss of mitochondrial energy production functions are metabolic and poorly treated hypertensive heart disease, environmental toxins, pesticides, heavy metals, or harmful drugs, poorly pulmonary or Rheumatic heart disease, surgical instrument injury, blunt chest trauma, in addition to environmental toxins or dangerous drugs, for example.

Myocardial Damage, colloquially known as "heart attack," is a portion of the myocardium that stops functioning. Myocardial damages may be “silent” and go undetected, or if the extent of the damage reaches a stage where the loss of functions could be a catastrophic event leading to hemodynamic deterioration and sudden death.

Myocarditis:

Myocarditis is inflammation of the heart muscle, and pericarditis is inflammation of the outer lining. In both cases, the immune system causes inflammation in response to an infection (i.e., viruses, fungi, bacteria) or some other trigger (e.g., too much Insulin used to treat Diabetes) or some other trigger (e.g., autoimmune disorders, drugs, vaccines, excessive Glucose intake, too much ultra-processed food intake, medications, environmental toxins, pesticides, Alcohol, etc.)

Pulmonary Heart Disease:

Pulmonary heart disease is a rare condition due to long-term tobacco smoking, COPD, Asthma, and long-term tobacco smoking, in which the heart cannot pump enough blood to the body. It usually begins with pulmonary hypertension or high blood pressure in the arteries inside the lungs. Pulmonary hypertension is a condition that specifically affects the arteries in the lungs. The causes can be long-term tobacco smoking, COPD, Asthma, and others.

Rheumatic Heart Disease:

Rheumatic heart disease occurs when the heart valves or other structures suffer damage due to rheumatic fever or other inflammatory/infectious/metabolic dysfunction factors. Heart valve damage may start shortly after an untreated or under-treated streptococcal infection, such as strep throat or scarlet fever. An immune response causes an inflammatory condition in the body, which can result in ongoing valve damage.

Congenital Heart Disease:

Congenital heart disease (CHD) is an issue with your heart’s abnormal structure formation present at birth. For example, one has a hole in your heart wall.

Issues with your blood vessels (too many or too few, blood flowing too slowly, to the wrong place, or in the wrong direction).

Problems with your heart valves that control blood flow.

Other inborn errors of metabolism, congenital or genetic defects such asHunter'ss Syndrome, Marfan syndrome, etc

Ventricular Hypertrophy: Left ventricular hypertrophy thickens the heart’s main pumping chamber (LV) wall. This thickening may result in blood pressure elevation within the heart and sometimes poor pumping action.

Right ventricular hypertrophy (RVH) is an abnormal enlargement or pathologic increase in the right ventricular muscle mass, which occurs as a maladaptive response to chronic pressure overload. RVH most commonly arises from chronic, severe lung disease.

Bilateral ventricular hypertrophy also exists and is measurable using MCG.

PHYSIOPATHOLOGICAL CONDITIONS: (For clinicians references and pattern recognition purposes only, not definitive diagnosis)

Myocardial Remodeling

Myocardial remodeling (REM) may be a damaging process characterized by gradual cardiac enlargement, cardiac dysfunction, and typical molecular changes. It is a universal phenomenon caused by many pathological conditions, the most common of which is myocardial infarction.

Athletes may assume they have “physiological remodeling.” However, other factors, which can be pathological,” may contribute to the impact.

Decreased myocardial compliance

The term compliance measures how difficult a heart chamber or the lumen of a blood vessel is to extend when it fills with blood. Physically, compliance (C) defines the volume change (ΔV) divided by the change in pressure (ΔP). Conditions such as long-term exposures to Ischemia or hypertension left unmanaged can lead to decreased compliance and, ultimately, heart failure, reduced ejection fractions,

Left ventricular filling is restricted when left ventricular compliance markedly decreases. When left ventricular compliance markedly decreases, stroke volume and cardiac output decrease, with signs and symptoms of low cardiac output. Pulmonary venous hypertension is associated with an obligatory increase in pulmonary artery pressure.

Increased myocardial compliance

Compliance is the inverse of stiffness and describes how easily the heart expands when blood fills up. As the ventricular filling increases, higher filling pressures develop nonlinearly for a given volume. Conditions such as acute myocardial infarction or myocarditis may lead to increased myocardial compliance. Please exercise Caution; caregivers must avoid risky actions such as sending patients to extraneous activities, including stress testing. Increased peripheral systemic resistance can lead to myocardium rupture and sudden death due to increased myocardial compliance.

Decreased ejection fraction

A clinically normal ejection fraction is between 55 and 70 percent. An EF from 41 to 49 hearts might be considered too low. It is important to note, however, that you can have an expected average ejection fraction measurement and still have heart failure. The condition is called HFpEF or heart failure with a preserved ejection fraction. MCG dynamically detects abnormal ejection fractions below 55 percent and then integrates the information into the overall severity scores functionally, physiologically, and mathematically. For nearly 50 percent of patients with clinical manifestations of heart failure, MCG is the ONLY test that can identify these patients, bar none.

Bradycardia:

Bradycardia is a slower-than-normal heart rate. A typical average adult resting heart rate is between 60 and 100 beats per minute (bpm). If you have bradycardia, your heart beats fewer than 60 times a minute. Bradycardia (heart rate 40 – 60 bpm) is typical when sleeping. However, medications, such as beta-blockers and other abnormal autonomic dysfunctions, can lead to abnormally lower heart rates than usual.

Tachycardia

Tachycardia means your heart beats faster than expected, usually more than 100 beats per minute. Some forms of tachycardia are typically “normal” in contrast; others can indicate a disease process or be life-threatening, especially when it occurs in conjunction with myocardial compliance issues, V-fib, synchronization, etc.

Acute Power Failure: MCG directly measures power output due to overall mitochondrial energy production dynamics. However, chronic conditions can also lead to persistent power failure, which may be harder to treat. Acute heart failure: Acute Power Failure is mainly due to mitochondrial failure or drastically reduced mitochondrial functions in energy production. Causes can be acute Ischemia, MI, metabolic dysfunctions, neurohormonal disorders, and acute stress. These are potentially reversible. However, Chronic conditions can also lead to persistent power failure, which may be harder to treat and reverse.

Global asynchrony (II < V5):

Global asynchrony in the heart is a delayed contraction of the right side of the heart. It can occur involving the right ventricle (RV) after a myocardial infarction (MI) due to a loss of contractility in the damaged heart muscle or hypertrophy due to hypertensive heart disease. This abnormal function can lead to LV global remodeling and dysfunction. Asynchrony can also relate to regional variation in the timing of diastolic events.

Global asynchrony (V5 < II) Global asynchrony in the heart is a delayed contraction of the left side of the heart. It can occur in the left ventricle (LV) after a myocardial infarction (MI) due to a loss of contractility in the infarct myocardial. This dysfunction can lead to LV global remodeling and dysfunction. Asynchrony can also lead to regional variation in the timing of diastolic events.

Localized Asynchrony

Mechanical cardiac asynchrony is a delayed contraction of specific myocardial regional segments; one part is out of the “phase” from its counterpart.

Local asynchrony (II < V5): Local asynchrony is a delayed contraction of the Right-Side Heart segment(s). It can occur in the left ventricle (LV) after a myocardial infarction (MI) due to a loss of contractility in the infarct myocardial. This dysfunction can lead to LV local remodeling and dysfunction. Asynchrony can also lead to regional variation in the timing of diastolic events.

Local asynchrony (V5 < II): Local asynchrony in the heart is a delayed contraction of the Left-Side Heart segment(s). It can occur in the left ventricle (LV) after a myocardial infarction (MI) due to a loss of contractility in the infarct myocardial. This dysfunction can lead to LV local remodeling and dysfunction. Asynchrony can also lead to regional variation in the timing of diastolic events.

My Comments:

The report will sound intimidating to you. MCG has learned these terms from years of data mining and deep learning from the legacy cardiology world. I decided to adopt these terms so we can communicate with the clinicians. Otherwise, those with the best and most brilliant minds will not easily understand and master the mathematical descriptions and digital output. The descriptions of each term are scary to many by design of legacy cardiology. However, please do not fall for it. They are designed to make you feel things are dire, but they are NOT. The good news is that there are simple things you can do for yourself to improve your mitochondrial functions and, thus, your metabolic health, and make more of them to reverse these trends. We have proven that.

Here they are:

1. Maintain a daily happy and positive attitude towards life, no matter how challenging and stressful. Be a happy warrior, and be resilient. Secure a solid six-hour uninterrupted sleep in total darkness.

2. To reach maximum heart rates, exercise regularly, practice high-intensity interval and weight training to build muscles, and do short bursts of rapid running three times a week to maintain maximum heart rate for a brief burst of time (~90-120 seconds).

3. Eat whole foods and avoid ultra-processed foods (seed oils, sugar, flour, and chemicals you do not recognize reading the ingredient lists). Learn the art of intermittent fasting and fasting to improve your mitochondrial functions and autophagy, and practice high-intensity interval training and short bursts of anaerobic exercises to grow your muscle strength and mass.

These will go a long way toward improving your metabolic health and adding new, fresh mitochondria to your cells. MCG can measure the improvements following these simple lifestyle optimization measures without adding more drugs to your burdened life.

There are plenty of examples where you can learn how lifestyle optimization can help you reverse and eliminate these scary terms in your following MCG report! We have witnessed these startling life-changing moments in patients treated “maximally” with PTCA/Stenting, Coronary Bypass Surgeries, and on multiple medications that benefit from these lifestyle measures. Initially, they were shocked to learn from their MCG reports that their “maximum treatments” were not enough, and then, they were relieved to discover that following these simple measures, the conditions improved measurably.

Most importantly, MCG‘s early detection capabilities will help you detect the earliest signs of mitochondrial dysfunction due to metabolic problems, allowing you to reverse these trends, prevent the progression of chronic diseases, stay healthier, and live longer without too much medical intervention for the overwhelming majority!

Help has arrived!

Here are some real-life examples:

For further questions, please get in touch with info@PremierHeart.com.

Our reality-based DML/NN system, built on shoestring budgets over the years, is anchored on a set of immutable and immortal mathematical principles reflecting the physiology of human biology supported with solid, vetted, reliable, trusted, and objective training data sets devoid of the unscalable backward legacy analog medicine’s biased “paid-expert-opinions-based” and against amoral dehumanizing “profit-only-motivated” agenda. We have repeatedly optimized the MCG tech platform reporting accuracy to save lives for the lowest costs. This 21st-century scalable forward-looking digital “100%-empirical-evidence-based” diagnostic information digital technology solution has taken us two and a half generations and decades to create; we have reliably solved critical medical diagnosis issues to support patient-centered decision-making to restore the lost trust and confidence in the failed 19th-century legacy “only-trusting-the-experts” medicine; in addition, we also have the monetization models figured out already.

Here are some examples of how we help people with “invisible” metabolic dysfunctions impacting their hearts:

And our journey to validate our claims in real-world clinical settings through blinded independent cream-of-the-crop ethical volunteering investigators without any industry standard, but corrupt quid pro quo:

Enjoy!

Humbly Yours, Joseph

As Arthur C. Clarke once said: "The only way to truly understand the limits of the possible is to push past them and explore the impossible." This philosophy guides our work at MCG, where we constantly push the boundaries of what is achievable in bioengineering and technology.

Joseph T. Shen, an independent, self-emancipated, recovered, freedom MD, became a self-taught BioCybernetic Systems Information Technology Engineer pioneer focused on developing Computational BioCybernetics and Lagrangian Mechanics Systems Engineering Deep-learning heart and brain-saving Intelligent Technologies.

Researcher and Technology Developer

Managing Member

Premier Heart, LLC

Premier Heart International, LLC

Premier Heart Japan, Inc.

Emergency MCG USA, Inc.

110 Main Street, Suite 201-88

Port Washington, NY 11050

Tel: 516-883-3383 ex 8102

Fax: 516-883-5812

Mobile: 516-603-6368 (Please alert me who you are with a text message first; please do not respond to unknown numbers. I WILL respond as soon as possible.)

Email: jtshenmd@premierheart.com (mailto:jtshenmd@premierheart.com)